Autism is a intricate neurological disorder which involves deviation at the stage of the system of central nervous. At this moment, the cause of it is still remain unclear most likely it’s still unknown. However, the inherited of genetics dysfunctions play a very main role as the suspect factors. The recent researches on the mice have been found that there is the connection between the abnormal behaviors and the genetics inherits. The scientists have been able to trigger the symptoms that are similar to those generated by an autism in human by deleting the PTEN gene in some part of the brain.

Even though, the researches can not give a clear explanation in the exact phenomenon which leads to the autism that developed in human. The suggestion which get from the results of the study might be related to the hippocampus, the area in the brain that leads the body to perform the abnormal action. Once the PTEN gene had been removed from the hippocampus, the behavioral patterns of the mouse are began to react like what in human autism. The abnormal behaviors are like the lack of social interactions, can not concentration, lost their temper.

From a physiological point of view

On the side of the physiological point, the thick nerves cells in an abnormal presented the dysfunctions of myelin, also the substance that surrounds the body’s neurons, which enabling the transmission of nervous impulses. The anatomical abnormal characteristic in mice with PTEN removed bring a big hope to those who willing to understand more in the autism in human. However, the medical scientists have been able to replicate the symptoms of autism in mice, new medical treatments can be tested.

The medical scientists who carry out the experiment were able to reveal many likeness between the actions of mice with the PTEN gene deleted also the behaviors of people with autism. The irregular mice involved in the experiment response a lot more interest in many objects than in other mice. They also became passive, and remote, avoiding any form of interaction with other mice. The major differences, was that unlike autistic in people, the abnormal mice didn’t show any signs of insistence on sameness or repetitive behaviors.

The results from the experiments show a strong connection between the PTEN gene and the pattern of the abnormal action. Nevertheless, the cause of the autism still not yet confirmed how it happened and what is the cause to blame.